American Society of Hirudotherapy

Study of the effect of leeching on plasma endothelin and soluble interleukin-2 receptor in patients with systemic lupus erythematosus

Cheng SP, Liu JL, Yuan J (2005) · Chinese Journal of Integrative Medicine · n=44

RCT evidence detailTrial reference
GRADE Very LowInsufficient evidenceCondition: Knee Osteoarthritis
Sample size of this trial compared with other Knee Osteoarthritis trialsWang H 2018264Lauche R 2025240Lauche R 2014237Farzali S 2025181Cui Y 2024144Andereya S 2008113Andereya S 2008113Sarbaev IS 201996Isik M 201790Cheng SP 200544
This trial (highlighted) by sample size alongside other indexed Knee Osteoarthritis trials. Larger trials generally carry more statistical weight.

Study Profile

Design
single-center, open-label, randomized controlled clinical trial of leeching as an add-on to conventional corticosteroid therapy in systemic lupus erythematosus (People's Hospital of Guizhou Province, China)
Sample size (n)
44
Intervention
Conventional corticosteroid treatment plus leeching intervention (n=24); plasma endothelin and soluble interleukin-2 receptor measured before and after treatment
Comparator
Conventional corticosteroid treatment alone (n=20)
Primary endpoint
Plasma endothelin (ET) and soluble interleukin-2 receptor (sIL-2R) concentrations before and after treatment
Primary result
Both groups showed significant improvement in plasma ET and sIL-2R after treatment (p<0.05); the leeching group demonstrated a significantly greater improvement than the conventional-only group (between-group p<0.05); authors concluded that adjunctive leeching improves the level of these biomarkers and may ameliorate renal tissue impairment in SLE
Follow-up duration
treatment course duration (specific weeks not reported in abstract)

Key Findings

  • Rare RCT-level evidence for leeching in a systemic autoimmune disease (SLE) context
  • Adjunctive leeching plus conventional corticosteroids produced significantly greater reduction in plasma endothelin and sIL-2R than corticosteroids alone
  • First trial-level data suggesting a biomarker-based anti-inflammatory mechanism for leech therapy in SLE
  • Chinese integrative-medicine context - leeching is part of the recognized traditional therapeutic toolkit
  • Mechanistic relevance to broader autoimmune indications, though the clinical relevance to organ outcomes remains uncertain

Limitations

  • Small sample (n=44) limits subgroup and sensitivity analyses
  • Single Chinese center with no Western replication of biomarker findings
  • Surrogate biomarker endpoints (ET, sIL-2R) rather than hard clinical outcomes (renal failure, mortality, flare frequency)
  • Open-label design with no sham control
  • Limited methodological detail on randomization, allocation concealment, and outcome assessment blinding

Clinical Implications

Cheng 2005 is the only PubMed-indexed RCT to test leeching in systemic lupus erythematosus. The biomarker improvements (plasma endothelin and sIL-2R) are mechanistically interesting and consistent with anti-inflammatory and vasodilatory properties of leech saliva. However, the trial does not address hard clinical outcomes (renal failure, mortality, lupus flare frequency) and should be cited only as a hypothesis-generating mechanistic signal rather than as evidence for routine clinical use. For US clinicians, leech therapy in SLE is not a recommended indication and the FDA 510(k) K040187 clearance does not cover this use. The trial is included in the registry for completeness of the international integrative-medicine evidence base.

Related Trials

This website provides educational information and does not constitute medical advice, diagnosis, or treatment recommendations. Medicinal leech therapy carries clinically meaningful risks and should be performed only by qualified clinicians under institutionally approved protocols. FDA 510(k) clearance for medicinal leeches is limited to specific indications; investigational and off-label discussions are labeled accordingly. For patient-specific guidance, consult a qualified healthcare provider.